Swiss-Prot entry

ID   GCR_RAT        STANDARD;      PRT;   795 AA.
AC   P06536; O08624; Q8R463; Q8R5J0;
DT   01-JAN-1988 (Rel. 06, Created)
DT   01-NOV-1995 (Rel. 32, Last sequence update)
DT   01-MAY-2005 (Rel. 47, Last annotation update)
DE   Glucocorticoid receptor (GR).
GN   Name=Nr3c1; Synonyms=Grl;
OS   Rattus norvegicus (Rat).
OC   Eukaryota; Metazoa; Chordata; Craniata; Vertebrata; Euteleostomi; 
OC   Mammalia; Eutheria; Euarchontoglires; Glires; Rodentia; Sciurognathi; 
OC   Muridae; Murinae; Rattus. 
OX   NCBI_TaxID=10116;
RN   [1]
RP   NUCLEOTIDE SEQUENCE.
RC   TISSUE=Hepatoma;
RX   MEDLINE=86272086; PubMed=3755378 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1016/0092-8674(86)90659-8;
RA   Miesfeld R., Rusconi S., Godowski P.J., Maler B.A., Okret S.,
RA   Wikstroem A.-C., Gustafsson J.-A., Yamamoto K.R.;
RT   "Genetic complementation of a glucocorticoid receptor deficiency by
RT   expression of cloned receptor cDNA.";
RL   Cell 46:389-399(1986).
RN   [2]
RP   NUCLEOTIDE SEQUENCE.
RA   Heeley R.P., Gill E., van Zutphen B.;
RT   "CAG repeat variation in the gene for rat glucocorticoid receptor: a
RT   study of allele frequencies in inbred and wild rat populations and of
RT   the steroid-binding properties of their polypeptides in vitro.";
RL   Submitted (APR-1997) to the EMBL/GenBank/DDBJ databases.
RN   [3]
RP   NUCLEOTIDE SEQUENCE, AND VARIANTS GLY-226 AND ASP-260.
RC   STRAIN=Brown Norway/Crl, and SHR/OlaIpcv;
RA   Pravenec M., Zidek V., Kostka V., Mlejnek P., Musilova A., Kren V.,
RA   Jansa P., Forejt J., Lezin E.S., Kurtz T.W.;
RT   "Genetic isolation of a QTL on chromosome 18 associated with salt
RT   sensitivity in spontaneously hypertensive rats.";
RL   Submitted (DEC-2001) to the EMBL/GenBank/DDBJ databases.
RN   [4]
RP   NUCLEOTIDE SEQUENCE OF 1-515.
RC   STRAIN=Sprague-Dawley; TISSUE=Prostate;
RX   MEDLINE=88067783; PubMed=3684608 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Chang C., Kokontis J., Chang C.T., Liao S.;
RT   "Cloning and sequence analysis of the rat ventral prostate
RT   glucocorticoid receptor cDNA.";
RL   Nucleic Acids Res. 15:9603-9603(1987).
RN   [5]
RP   MUTAGENESIS OF ZINC FINGER.
RX   MEDLINE=89052664; PubMed=3191912 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Severne Y., Wieland S., Schaffner W., Rusconi S.;
RT   "Metal binding 'finger' structures in the glucocorticoid receptor
RT   defined by site-directed mutagenesis.";
RL   EMBO J. 7:2503-2508(1988).
RN   [6]
RP   GLUCOCORTICOID-MEDIATED DOWN-REGULATION.
RX   MEDLINE=89112207; PubMed=3216865 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Dong Y., Poellinger L., Gustafsson J.-A., Okret S.;
RT   "Regulation of glucocorticoid receptor expression: evidence for
RT   transcriptional and posttranslational mechanisms.";
RL   Mol. Endocrinol. 2:1256-1264(1988).
RN   [7]
RP   MUTAGENESIS OF CYS-656.
RX   MEDLINE=92042128; PubMed=1939229 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Chakraborti P.K., Garabedian M.J., Yamamoto K.R., Simons S.S. Jr.;
RT   "Creation of 'super' glucocorticoid receptors by point mutations in
RT   the steroid binding domain.";
RL   J. Biol. Chem. 266:22075-22078(1991).
RN   [8]
RP   MUTAGENESIS OF ZINC FINGER.
RX   MEDLINE=93195921; PubMed=8450530 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Zandi E., Galli I., Doebbeling U., Rusconi S.;
RT   "Zinc finger mutations that alter domain interactions in the
RT   glucocorticoid receptor.";
RL   J. Mol. Biol. 230:124-136(1993).
RN   [9]
RP   REVIEW ON MUTAGENESIS.
RX   MEDLINE=94249031; PubMed=8191545 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1016/0039-128X(94)90093-0;
RA   Lanz R.B., Hug M., Gola M., Tallone T., Wieland S., Rusconi S.;
RT   "Active, interactive, and inactive steroid receptor mutants.";
RL   Steroids 59:148-152(1994).
RN   [10]
RP   HOMODIMERIZATION, HETERODIMERIZATION WITH NR3C2, AND MUTAGENESIS OF
RP   ASP-481.
RX   MEDLINE=96109290; PubMed=8618925 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Liu W., Wang J., Sauter N.K., Pearce D.;
RT   "Steroid receptor heterodimerization demonstrated in vitro and in
RT   vivo.";
RL   Proc. Natl. Acad. Sci. U.S.A. 92:12480-12484(1995).
RN   [11]
RP   INTERACTION WITH NCOA2.
RX   MEDLINE=97265407; PubMed=9111344 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Hong H., Kohli K., Garabedian M.J., Stallcup M.R.;
RT   "GRIP1, a transcriptional coactivator for the AF-2 transactivation
RT   domain of steroid, thyroid, retinoid, and vitamin D receptors.";
RL   Mol. Cell. Biol. 17:2735-2744(1997).
RN   [12]
RP   PHOSPHORYLATION SITES THR-171; SER-224; SER-232 AND SER-246.
RX   MEDLINE=98269094; PubMed=9603939 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1074/jbc.273.23.14315;
RA   Rogatsky I., Waase C.L.M., Garabedian M.J.;
RT   "Phosphorylation and inhibition of rat glucocorticoid receptor
RT   transcriptional activation by glycogen synthase kinase-3 (GSK-3).
RT   Species-specific differences between human and rat glucocorticoid
RT   receptor signaling as revealed through GSK-3 phosphorylation.";
RL   J. Biol. Chem. 273:14315-14321(1998).
RN   [13]
RP   INTERACTION WITH NRIP1.
RX   MEDLINE=99292791; PubMed=10364267 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1074/jbc.274.25.18121;
RA   Subramaniam N., Treuter E., Okret S.;
RT   "Receptor interacting protein RIP140 inhibits both positive and
RT   negative gene regulation by glucocorticoids.";
RL   J. Biol. Chem. 274:18121-18127(1999).
RN   [14]
RP   INTERACTION WITH POU2F1 AND POU2F2, AND MUTAGENESIS OF CYS-500 AND
RP   LEU-501.
RX   MEDLINE=99410400; PubMed=10480874 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1074/jbc.274.38.26713;
RA   Prefontaine G.G., Walther R., Giffin W., Lemieux M.E., Pope L.,
RA   Hache R.J.G.;
RT   "Selective binding of steroid hormone receptors to octamer
RT   transcription factors determines transcriptional synergism at the
RT   mouse mammary tumor virus promoter.";
RL   J. Biol. Chem. 274:26713-26719(1999).
RN   [15]
RP   INTERACTION WITH NCOA6.
RX   MEDLINE=20325329; PubMed=10866662 [NCBI, ExPASy, EBI, Israel, Japan];
RX   DOI=10.1128/MCB.20.14.5048-5063.2000;
RA   Mahajan M.A., Samuels H.H.;
RT   "A new family of nuclear receptor coregulators that integrates nuclear
RT   receptor signaling through CBP.";
RL   Mol. Cell. Biol. 20:5048-5063(2000).
RN   [16]
RP   HOMODIMERIZATION, HETERODIMERIZATION WITH NR3C2, AND MUTAGENESIS OF
RP   LEU-501.
RX   MEDLINE=21216736; PubMed=11278286 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1074/jbc.M005363200;
RA   Ou X.-M., Storring J.M., Kushwaha N., Albert P.R.;
RT   "Heterodimerization of mineralocorticoid and glucocorticoid receptors
RT   at a novel negative response element of the 5-HT1A receptor gene.";
RL   J. Biol. Chem. 276:14299-14307(2001).
RN   [17]
RP   ALTERNATIVE INITIATION, AND MUTAGENESIS OF MET-1 AND MET-28.
RX   MEDLINE=21329577; PubMed=11435610 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1210/me.15.7.1093;
RA   Yudt M.R., Cidlowski J.A.;
RT   "Molecular identification and characterization of A and B forms of the
RT   glucocorticoid receptor.";
RL   Mol. Endocrinol. 15:1093-1103(2001).
RN   [18]
RP   INTERACTION WITH NCOA1, AND MUTAGENESIS OF GLU-773.
RX   MEDLINE=22113105; PubMed=12118039 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1074/jbc.M204013200;
RA   Kucera T., Waltner-Law M., Scott D.K., Prasad R., Granner D.K.;
RT   "A point mutation of the AF2 transactivation domain of the
RT   glucocorticoid receptor disrupts its interaction with steroid receptor
RT   coactivator 1.";
RL   J. Biol. Chem. 277:26098-26102(2002).
RN   [19]
RP   X-RAY CRYSTALLOGRAPHY (2.5 ANGSTROMS) OF 434-525.
RX   MEDLINE=91326070; PubMed=1865905 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1038/352497a0;
RA   Luisi B.F., Xu W.X., Otwinowski Z., Freedman L.P., Yamamoto K.R.,
RA   Sigler P.B.;
RT   "Crystallographic analysis of the interaction of the glucocorticoid
RT   receptor with DNA.";
RL   Nature 352:497-505(1991).
RN   [20]
RP   STRUCTURE BY NMR OF 440-510.
RX   MEDLINE=90319784; PubMed=2115209 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Haerd T., Kellenbach E., Boelens R., Maler B.A., Dahlman K.,
RA   Freedman L.P., Carlstedt-Duke J., Yamamoto K.R., Gustafsson J.-A.,
RA   Kaptein R.;
RT   "Solution structure of the glucocorticoid receptor DNA-binding
RT   domain.";
RL   Science 249:157-160(1990).
RN   [21]
RP   STRUCTURE BY NMR OF 440-525.
RX   MEDLINE=92089066; PubMed=1751485 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Remerowski M.L., Kellenbach E., Boelens R., van der Marel A.,
RA   van Boom J.H., Maler B.A., Yamamoto K.R., Kaptein R.;
RT   "1H NMR studies of DNA recognition by the glucocorticoid receptor:
RT   complex of the DNA binding domain with a half-site response element.";
RL   Biochemistry 30:11620-11624(1991).
RN   [22]
RP   STRUCTURE BY NMR OF 440-525.
RX   MEDLINE=92038072; PubMed=1936288 [NCBI, ExPASy, EBI, Israel, Japan]; DOI=10.1016/0014-5793(91)81322-Y;
RA   Kellenbach E., Maler B.A., Yamamoto K.R., Boelens R., Kaptein R.;
RT   "Identification of the metal coordinating residues in the DNA binding
RT   domain of the glucocorticoid receptor by 113Cd-1H heteronuclear NMR
RT   spectroscopy.";
RL   FEBS Lett. 291:367-370(1991).
RN   [23]
RP   STRUCTURE BY NMR OF 439-510.
RX   MEDLINE=94079877; PubMed=8257681 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Baumann H., Paulsen K., Kovacs H., Berglund H., Wright A.P.H.,
RA   Gustafsson J.-A., Haerd T.;
RT   "Refined solution structure of the glucocorticoid receptor DNA-binding
RT   domain.";
RL   Biochemistry 32:13463-13471(1993).
RN   [24]
RP   POLYMORPHISM OF POLY-GLN REGION.
RX   MEDLINE=93261843; PubMed=8493115 [NCBI, ExPASy, EBI, Israel, Japan];
RA   Gearing K.L., Gustafsson J.-A., Okret S.;
RT   "Heterogeneity in the polyglutamine tract of the glucocorticoid
RT   receptor from different rat strains.";
RL   Nucleic Acids Res. 21:2014-2014(1993).
CC   -!- FUNCTION: Receptor for glucocorticoids (GC). Has a dual mode of
CC       action: as a transcription factor that binds to glucocorticoid
CC       response elements (GRE) and as a modulator of other transcription
CC       factors. Affects inflammatory responses, cellular proliferation
CC       and differentiation in target tissues.
CC   -!- SUBUNIT: Heteromultimeric cytoplasmic complex with HSP90, HSP70,
CC       and FKBP5 or another immunophilin, or the immunophilin homolog
CC       PPP5C. Upon ligand binding FKBP5 dissociates from the complex and
CC       FKBP4 takes its place, thereby linking the complex to dynein and
CC       mediating transport to the nucleus, where the complex dissociates.
CC       Binds to DNA as a homodimer, and as heterodimer with NR3C2 or the
CC       retinoid X receptor. Binds STAT5A and STAT5B homodimers and
CC       heterodimers. Interacts with NRIP1, TGFB1I1, POU2F1, POU2F2 and
CC       TRIM28. Interacts with several coactivator complexes, including
CC       the SMARCA4 complex, CREBBP/EP300, TADA2L (Ada complex) and p160
CC       coactivators such as NCOA2 and NCOA6. Interaction with BAG1
CC       inhibits transactivation (By similarity).
CC   -!- SUBCELLULAR LOCATION: Cytoplasmic in the absence of ligand;
CC       nuclear after ligand-binding (By similarity).
CC   -!- ALTERNATIVE PRODUCTS:
CC       Event=Alternative initiation;
CC         Comment=2 isoforms, A (shown here) and B, are produced by
CC         alternative initiation at Met-1 and Met-27;
CC   -!- DOMAIN: Composed of three domains: a modulating N-terminal domain,
CC       a DNA-binding domain and a C-terminal steroid-binding domain.
CC   -!- PTM: Increased proteasome-mediated degradation in response to
CC       glucocorticoids.
CC   -!- PTM: Phosphorylated in the absence of hormone; becomes
CC       hyperphosphorylated in the presence of glucocorticoids.
CC   -!- PTM: Sumoylated; this reduces transcription transactivation (By
CC       similarity).
CC   -!- SIMILARITY: Belongs to the nuclear hormone receptor family. NR3
CC       subfamily.
CC   -!- SIMILARITY: Contains 1 nuclear receptor DNA-binding domain.
CC   --------------------------------------------------------------------------
CC   This Swiss-Prot entry is copyright. It is produced through a collaboration
CC   between  the Swiss Institute of Bioinformatics  and the  EMBL outstation -
CC   the European Bioinformatics Institute.  There are no  restrictions on  its
CC   use as long as its content is in no way modified and this statement is not
CC   removed.
CC   --------------------------------------------------------------------------
DR   EMBL; M14053; AAA41203.1; -. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; Y12264; CAA72938.1; -. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; AY066016; AAL66772.2; -. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; AF455050; AAL78956.1; -. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; Y00489; CAA68545.1; -. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; X69666; -; NOT_ANNOTATED_CDS. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; X69667; -; NOT_ANNOTATED_CDS. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; X69668; -; NOT_ANNOTATED_CDS. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; X69669; -; NOT_ANNOTATED_CDS. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   EMBL; X69670; -; NOT_ANNOTATED_CDS. [EMBL / GenBank / DDBJ] [CoDingSequence]
DR   PIR; A24194; QRRTG.
DR   PDB; 1GDC; NMR; @=439-510. [ExPASy / RCSB]
DR   PDB; 1GLU; X-ray; A/B=434-514. [ExPASy / RCSB]
DR   PDB; 1LAT; X-ray; A/B=434-515. [ExPASy / RCSB]
DR   PDB; 1R4O; X-ray; A/B=434-525. [ExPASy / RCSB]
DR   PDB; 1R4R; X-ray; A/B=434-525. [ExPASy / RCSB]
DR   PDB; 1RGD; NMR; @=440-510. [ExPASy / RCSB]
DR   PDB; 2GDA; NMR; @=439-510. [ExPASy / RCSB]
DR   SMR; P06536; 541-795.
DR   TRANSFAC; T00333; -.
DR   Ensembl; ENSRNOG00000014096; Rattus_norvegicus
DR   RGD; 2741; Nr3c1.
DR   InterPro; IPR001409; Glcrtcd_receptor.
DR   InterPro; IPR000536; Hrmon_recept_lig.
DR   InterPro; IPR001723; Stdhrmn_receptor.
DR   InterPro; IPR008946; Str_ncl_receptor.
DR   InterPro; IPR001628; Znf_C4steroid.
DR   InterPro; Graphical view of domain structure.
DR   Pfam; PF02155; GCR; 1.
DR   Pfam; PF00104; Hormone_recep; 1.
DR   Pfam; PF00105; zf-C4; 1.
DR   Pfam; Graphical view of domain structure.
DR   PRINTS; PR00528; GLCORTICOIDR.
DR   PRINTS; PR00398; STRDHORMONER.
DR   PRINTS; PR00047; STROIDFINGER.
DR   ProDom; PD000035; Znf_C4steroid; 1.
DR   ProDom [Domain structure / List of seq. sharing at least 1 domain ]
DR   SMART; SM00430; HOLI; 1.
DR   SMART; SM00399; ZnF_C4; 1.
DR   PROSITE; PS00031; NUCLEAR_REC_DBD_1; 1.
DR   PROSITE; PS51030; NUCLEAR_REC_DBD_2; 1.
DR   CMR; P06536.
DR   HOVERGEN [Family / Alignment / Tree]
DR   BLOCKS; P06536.
DR   ProtoNet; P06536.
DR   ProtoMap; P06536.
DR   PRESAGE; P06536.
DR   DIP; P06536.
DR   ModBase; P06536.
DR   SWISS-2DPAGE; GET REGION ON 2D PAGE.
KW   3D-structure; Alternative initiation; DNA-binding; Nuclear protein;
KW   Phosphorylation; Polymorphism; Receptor; Steroid-binding;
KW   Trans-acting factor; Transcription; Transcription regulation;
KW   Ubl conjugation; Zinc-finger.
FT   CHAIN         1    795       Glucocorticoid receptor, A-type isoforms.
FT   CHAIN        28    795       Glucocorticoid receptor, B-type isoforms.
FT   INIT_MET     28     28       For B-type isoforms.
FT   DOMAIN        1    439       Modulating.
FT   DOMAIN      419    438       Glu/Ser/Pro/Thr-rich (PEST region)
FT                                (Potential).
FT   DNA_BIND    440    505       Nuclear receptor-type.
FT   ZN_FING     440    460       C4-type.
FT   ZN_FING     476    500       C4-type.
FT   DOMAIN      506    546       Hinge.
FT   DOMAIN      547    795       Steroid-binding.
FT   DOMAIN       75     97       Poly-Gln.
FT   MOD_RES     134    134       Phosphoserine (By similarity).
FT   MOD_RES     171    171       Phosphothreonine.
FT   MOD_RES     162    162       Phosphoserine (By similarity).
FT   MOD_RES     224    224       Phosphoserine.
FT   MOD_RES     232    232       Phosphoserine.
FT   MOD_RES     246    246       Phosphoserine.
FT   MOD_RES     327    327       Phosphoserine (By similarity).
FT   VARIANT      77     77       Missing (in strain Brown Norway/Crl).
FT   VARIANT      78     79       Missing (in strain SHR/OlaIpcv).
FT   VARIANT      83     96       Missing (in strain Sprague-Dawley).
FT   VARIANT     226    226       S -> G (in strain SHR/OlaIpcv and strain
FT                                Brown Norway/Crl).
FT   VARIANT     260    260       N -> D (in strain SHR/OlaIpcv and strain
FT                                Brown Norway/Crl).
FT   MUTAGEN       1      1       M->T: Abolishes expression of A-type
FT                                isoforms.
FT   MUTAGEN      28     28       M->T: Abolishes expression of B-type
FT                                isoforms.
FT   MUTAGEN     481    481       D->R: Disrupts dimerization and decreases
FT                                transcription transactivation.
FT   MUTAGEN     500    500       C->Y: Abolishes interaction with POU2F2.
FT   MUTAGEN     501    501       L->P: Abrogates DNA-binding and
FT                                transcription transactivation. Abolishes
FT                                interaction with POU2F1 and POU2F2.
FT   MUTAGEN     656    656       C->S: Strongly increases affinity for
FT                                dexamethasone.
FT   MUTAGEN     773    773       E->A: Abolishes interaction with NCOA1
FT                                and reduces transcription
FT                                transactivation; when associated with S-
FT                                656.
FT   CONFLICT     98     98       D -> G (in Ref. 1).
FT   CONFLICT    345    345       S -> T (in Ref. 4).
FT   CONFLICT    600    600       L -> P (in Ref. 2 and 3).
FT   CONFLICT    602    602       L -> F (in Ref. 3).
FT   STRAND      439    439       
FT   TURN        441    443       
FT   STRAND      446    446       
FT   STRAND      449    450       
FT   STRAND      455    456       
FT   HELIX       458    469       
FT   TURN        486    487       
FT   HELIX       488    490       
FT   TURN        491    491       
FT   HELIX       493    503       
FT   TURN        504    504       
SQ   SEQUENCE   795 AA;  87556 MW;  9C9DE0B1D6724845 CRC64;
     MDSKESLAPP GRDEVPGSLL GQGRGSVMDF YKSLRGGATV KVSASSPSVA AASQADSKQQ
     RILLDFSKGS TSNVQQRQQQ QQQQQQQQQQ QQQQQQPDLS KAVSLSMGLY MGETETKVMG
     NDLGYPQQGQ LGLSSGETDF RLLEESIANL NRSTSVPENP KSSTSATGCA TPTEKEFPKT
     HSDASSEQQN RKSQTGTNGG SVKLYPTDQS TFDLLKDLEF SAGSPSKDTN ESPWRSDLLI
     DENLLSPLAG EDDPFLLEGN TNEDCKPLIL PDTKPKIKDT GDTILSSPSS VALPQVKTEK
     DDFIELCTPG VIKQEKLGPV YCQASFSGTN IIGNKMSAIS VHGVSTSGGQ MYHYDMNTAS
     LSQQQDQKPV FNVIPPIPVG SENWNRCQGS GEDSLTSLGA LNFPGRSVFS NGYSSPGMRP
     DVSSPPSSSS AATGPPPKLC LVCSDEASGC HYGVLTCGSC KVFFKRAVEG QHNYLCAGRN
     DCIIDKIRRK NCPACRYRKC LQAGMNLEAR KTKKKIKGIQ QATAGVSQDT SENPNKTIVP
     AALPQLTPTL VSLLEVIEPE VLYAGYDSSV PDSAWRIMTT LNMLGGRQVI AAVKWAKAIL
     GLRNLHLDDQ MTLLQYSWMF LMAFALGWRS YRQSSGNLLC FAPDLIINEQ RMSLPCMYDQ
     CKHMLFVSSE LQRLQVSYEE YLCMKTLLLL SSVPKEGLKS QELFDEIRMT YIKELGKAIV
     KREGNSSQNW QRFYQLTKLL DSMHEVVENL LTYCFQTFLD KTMSIEFPEM LAEIITNQIP
     KYSNGNIKKL LFHQK
//